the Therapy and Prevention of Osteoporosis
By treating only the physical
symptoms to cure an illness or by trying to prevent it, targeting only
the symptoms, we will very likely neither cure it nor prevent it. This
knowledge is not new, but in regard to osteoporosis no current therapeutic
approach is taking it to heart. Neither the diagnosis nor the therapy of
osteoporosis are attempting significantly more than damage control. Prevention
is a mere lip service to the «customer» because the courage
to really beat this disease is nowhere to be found.
But then, based on the slender
body of knowledge available today, how could it be any different. None
of the traditional concepts of bone remodeling are conclusive and therefore
none of the therapeutic approaches based upon them can be convincing. On
the other hand, the commercial interests involved have no incentive to
change the lucrative status quo of osteoporosis prevention by «crop
dusting» women with estrogen.
This is the background on
which the author developed his
Program for Preventing and Healing of
Osteoporosis. This program teaches the reader (intentionally never
called «patient») to develop an intimate relationship with
his/her body and especially with her bones. S/he will learn to experience
the body as a partner with whom s/he can communicate -- without medical
jargon as a prerequisite. To this end, along with a nutritional approach,
avoiding stress and guilt, a program of movement and exercise is developed
which is to be understood as a practical and equitable communication with
the body. This in turn is built into a focussed therapy of relaxation and
The amazing aspect of this
program for preventing and healing osteoporosis is that is uses three methods
which are not at all new. In combination, however, they produce sensational
results: The development of an informed and responsible body consciousness,
of sane and self-loving nutritional habits and most importantly, movement
and exercise as a way to communicate. To prepare for this communcation,
the author explains how bone lives, how and when the loss of bone mass
really occurs, and he describes in detail the three columns of their program.
Finally, women get to tell their stories, women who themselves experienced
such a fundamental change. «My body is no longer foreign territory,
no longer a mere burden,» tells a woman who only half a year
ago felt paralyzed, caught between the fear of cancerogenic estrogens and
the threat of premature invalidity. «Today, I like my body and
I think it likes me, too. My daily exercises I do because they give me
joy and make me feel alive. By now, I have almost forgotten that I originally
learned them as part of an osteoporosis prevention program.»
This «Revolution in
the Therapy and Prevention of Osteoporosis» is based on a . . .
REVOLUTION IN THE
. . . of the endocrinal
Systems of hormonal regulation
are subject to influences of the central nervous system, of the psyche,
and of daily rhythms. These are known facts, but when it comes to human
bone metabolism they seem to be largely ignored. In relation to the bone,
daily rhythms are especially significant because of an increased nocturnal
absorption of bone tissue. This in turn creates an inevitable connection
to other events in the body which are subject to rhythms of day and night
or light and dark, predominantly, the production of serotonine and melatonine
and syndromes like winter depression. There can be no doubt, moods like
doom or cheer on life and love must have a ruling influence not only on
vegetative functions (blood pressure, breathing, pulse etc.) but also on
bone metabolism and bone resorption.
It is also a proven fact
that one and the same level of a hormonal substance shows a different effect
depending on whether it is the body’s «original» or if it has
been substituted. An example is thyreoid hormone. A serum thyroxin identical
to the natural level ravages bones mass -- but only when it has been substituted.
Interleukin-I is a
similar case. It is produced within the immune system and within the ovaries.
It stimulates proteolytic agents (collagenases) and therefore serves as
one of the main drives of the immune system (to «digest» antigens).
A secondary purpose of these collagenases is to dissolve the bone tissue
matrix. Exessive amounts of interleukin-I are suppressed by estradiol produced
in the ovaries. This is the reason estrogens are being hailed guardians
of the bone mass. If, however, estrogens are substituted after the ovaries
have ceased their hormonal production (menopause), the interleukin is suppresed
without compensation by the original interleukin from the ovaries. Consequently,
the immune system will slowly but surely get weakened.
. . . of the gender-specific
difference in bone mass loss:
Based on the known and undisputed
it necessarily follows that
bone remodeling takes place
on the surfaces of the bone,
the spongiosa contains by far
the largest portion of the bone surface,
the blood cell producing bone
marrow lies within the spongiosa, and
women run an increased risk
of developing osteoporosis once their menstruation has ceased and the demand
for increased blood production has thus ended,
. . . of bone remodeling:
at least one of the gender-specific
differences in bone metabolism is to be found in the higher percentage
of spongiosa in female bones and
the increased risk of women
for developing osteoporosis must be attributed to the impoverished nutrition
of the spongiosa after the menopause (due to the decreased demand for production
of new blood cells).
Traditional models of bone
remodeling were based on the notion of 4 steps (1. activation of osteoclast
precursors; 2. resorption; 3. reversal; 4. rebuilding) and on so-called
that is teams, in which osteoclasts and osteoblasts work together at a
fixed ratio. These two models, however, could only coexist in the idealistic
world of perfect harmony. As soon as we look for an explanation for a disbalance
between resorption and apposition (i.e. a loss of bone mass ð osteoporosis)
these models crash. Even inventing the new term «de-coupling»
(of osteoclasts from osteoblasts) could not convincingly save the old model.
How could it be any different if even different biopsy techniques can produce
diametrically opposite results.
The missing step was
to separate the construction of the bone matrix tissue from its mineralisation.
Traditionally, both were attributed to the osteoblasts within step 4. But
only by splitting up this step and reassigning responsibility to two different
entities osteoporosis can be explained and made transparent.
. . . of diagnosis
and patient history:
Alienation from ones own
body is the underlying cause of osteoporosis. To recognize this means no
finger pointing, as it is so popular in new-age psychology and medicine.
Instead, the facts discovered in taking the patient history can become
a key for diagnosis as well as therapy: «How would I react if
I were my own body and had to function for a boss with so little self worth?»
or «Does it make sense to become strong for someone who’s going
to give up on himself sooner or later anyway?»
REVOLUTION in THERAPY
This approach makes exercise
and specific load inducement into a sustainable means of communication,
integrating and including bone cells into the body system as a whole. As
individuals, each of us can be part of this communication provided we commit
ourselves to an equitable partnership with our body.
Specifically, this means
that, by using two known and undisputed processes (formation of callouses
& remodeling) even in places where a structural loss of bone tissue
has occured, such damage can first be repaired and then brought up to its
best possible structural quality.